![]() ![]() Taurine is a very common part of energy drinks, but its effects are definitely limited. Revitalise your body & order your pack of ENERGY now Buy now What is taurine good for? This article aims to inform you on all the reasons taurine is used, best ways to get enough of it through regular diet and which taurine supplement you should use if you do decide you want supplementation. The amounts of taurine in energy drinks can be quite high, but its effects are limited. While taurine is ever present in most energy and pre workout drinks, the fact is that it is not such an important ingredient in these, and its effects are not felt nearly as much as those of caffeine or other stimulants used in such products. Unlike caffeine and most other ingredients that you will find in such products, taurine is not a stimulant but rather a sedative that can also be used to reduce feelings of anxiety and similar disorders. TauT expression may be involved in the expression of various genes regulated by taurine supplementation.Īdipogenesis Brown adipose tissue Taurine Taurine transporter White adipose tissue.A conditionally essential amino acid that occurs naturally in our bodies but needs to be supplemented on some occasions, taurine is one of the most common ingredients in various energy drinks and other similar products whose goal is to promote energy and wakefulness. Long-term taurine supplementation causes weight loss, most likely by inhibiting adipogenesis in WAT. Consistently, the expression of taurine transporter (TauT) and adipocyte-specific genes such as adiponectin, leptin, and IL-6 was regulated in a similar pattern by taurine supplementation. Taurine supplementation showed the downregulation of these genes in WAT but not in BAT or muscle. The expression of adipogenesis-related genes such as PPAR-α, PPAR-γ, C/EBP-α, C/EBP-β, and AP2 increased in BAT and WAT, but not in muscle tissue. HFD-fed mice did not show significant difference of oxygen consumption (VO 2), energy expenditure (EE), respiratory exchange rate (RER), and locomotive activity compared with those of normal chow diet fed mice. Taurine supplementation showed the trend of anti-obesity effect in ICR mice fed an HFD for 28 weeks. Interscapular brown fat (BAT), inguinal white fat tissue (WAT), and quadriceps muscle were analyzed and compared to assess the change of gene expression related to adipogenesis. Metabolic, behavioral, and physiological monitoring were carried out using PhenoMaster at 28 weeks. Three groups of male ICR mice were fed a normal chow diet, a high-fat diet (HFD), or an HFD supplemented with 2% taurine in drinking water for 28 weeks. ![]() study was conducted to evaluate the anti-obesity effects of long-term taurine supplementation in a mild obese ICR mouse model and to study the mechanism by which taurine induces weight loss. 8 Department of Physiology, School of Medicine, Kyung Hee University, Seoul, 02447, Republic of Korea.3 Department of Clinical Pharmacology and Therapeutics, School of Medicine, Kyung Hee University, Seoul, 02447, Republic of Korea. 2 East-West Bone and Joint Disease Research Institute, Kyung Hee University Hospital at Gangdong, Gandong-gu, Seoul, Republic of Korea. 1 Department of Clinical Pharmacology and Therapeutics, School of Medicine, Kyung Hee University, Seoul, 02447, Republic of Korea. 7 East-West Bone and Joint Disease Research Institute, Kyung Hee University Hospital at Gangdong, Gandong-gu, Seoul, Republic of Korea.6 Interdisciplinary Program for Bioinformatics, Program for Cancer Biology and BIO-MAX/N-Bio Institute, Seoul National University, Seoul, 08826, Republic of Korea.5 Laboratory of Developmental Biology and Genomics, Research Institute for Veterinary Science, BK21 PLUS Program for Creative Veterinary Science Research, College of Veterinary Medicine, and Korea Mouse Phenotyping Center (KMPC), Seoul National University, Seoul, 08826, Republic of Korea.4 Severance Biomedical Science Institute Gangnam Severance Hospital BK21 PLUS Project for Medical Science, Yonsei University College of Medicine, Seoul, Republic of Korea.1 Department of Clinical Pharmacology and Therapeutics, School of Medicine, Kyung Hee University, Seoul, 02447, Republic of Korea. ![]()
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